46 Results for: "Trimethylamine"

Supplier: Thermo Fisher Scientific

Trimethylamine 1 M in THF, AcroSeal®

Supplier: Thermo Fisher Scientific

Trimethylamine 1 M in THF

Supplier: Merck

Trimethylamine 40% (w/w) in aqueous solution for synthesis, Sigma-Aldrich®

Supplier: Thermo Fisher Scientific

Trimethylamine 33% (w/w) 4.2 M in ethanol, pure

Supplier: Thermo Fisher Scientific

Trimethylamine 33% (w/w) in ethanol denatured with 2% cyclohexane

Supplier: Thermo Fisher Scientific

Trimethylamine 50% (w/w) 7.3 M in aqueous solution, pure

Supplier: Thermo Fisher Scientific

Trimethylamine 45% (w/w) in aqueous solution

Supplier: Thermo Fisher Scientific

Borane trimethylamine complex 97%

Supplier: Thermo Fisher Scientific

Sulphur trioxide trimethylamine complex 95%

Supplier: Thermo Fisher Scientific

Trimethylamine-N-oxide dihydrate 98%

Supplier: Thermo Fisher Scientific

Borane trimethylamine complex 97%

Supplier: Molekula

Trimethylamine-N-oxide dihydrate

Supplier: Apollo Scientific

Sulphur trioxide trimethylamine complex 95%

Supplier: Thermo Fisher Scientific

Trimethylamine-N-oxide dihydrate ≥98%

Supplier: Thermo Fisher Scientific

Sulphur trioxide trimethylamine complex 95%

Supplier: Merck

Organic Standard, Trimethylamine hydrochloride, ≥98,5% (GC)

Supplier: VWR Chemicals

Amine Standard, Trimethylamine, 1000 mg/L, VWR®, Matrix: H₂O, Application: Ion chromatography standards

Sale

Supplier: Honeywell Chemicals

Certified Reference Material (CRM)

Supplier: Cayman Chemical

Trimethylamine N-oxide 1 * 5 g

Supplier: Cayman Chemical

Trimethylamine N-oxide 1 * 1 g

Supplier: Apollo Scientific

Trimethylamine N-Oxide Anhydrous 25g pack 1 * 25 g

Supplier: Cayman Chemical

Trimethylamine N-oxide 1 * 500 mg

Supplier: Apollo Scientific

Trimethylamine N-Oxide Anhydrous 1g pack 1 * 1 g

Supplier: Apollo Scientific

Trimethylamine N-Oxide Anhydrous 5g pack 1 * 5 g

Supplier: Bioss

The Flavin containing monooxygenase family consists of five gene products, FMO1-5, that are major enzymatic oxidants involved in the metabolism of various therapeutics. Located in the liver, FMO3 is a hepatic microsomal enzyme that oxygenates soft nucleophiles such as secondary and tertiary amines. Through its N-oxygenase capabilities, FMO3 acts on a variety of xenobiotics to catalyze oxidative digestion. Defects in the FMO3 gene are the primary cause of trimethylaminuria (TMAuria), an inborn error of metabolism associated with a fishy body odor emitting from sweat, urine and breath. Genetic mutations in FMO3 lead to the N-oxidation of amino-trimethylamine derived from food products, thus producing the malodor associated with TMAuria.

Supplier: Bioss

The Flavin containing monooxygenase family consists of five gene products, FMO1-5, that are major enzymatic oxidants involved in the metabolism of various therapeutics. Located in the liver, FMO3 is a hepatic microsomal enzyme that oxygenates soft nucleophiles such as secondary and tertiary amines. Through its N-oxygenase capabilities, FMO3 acts on a variety of xenobiotics to catalyze oxidative digestion. Defects in the FMO3 gene are the primary cause of trimethylaminuria (TMAuria), an inborn error of metabolism associated with a fishy body odor emitting from sweat, urine and breath. Genetic mutations in FMO3 lead to the N-oxidation of amino-trimethylamine derived from food products, thus producing the malodor associated with TMAuria.

Supplier: Bioss

The Flavin containing monooxygenase family consists of five gene products, FMO1-5, that are major enzymatic oxidants involved in the metabolism of various therapeutics. Located in the liver, FMO3 is a hepatic microsomal enzyme that oxygenates soft nucleophiles such as secondary and tertiary amines. Through its N-oxygenase capabilities, FMO3 acts on a variety of xenobiotics to catalyze oxidative digestion. Defects in the FMO3 gene are the primary cause of trimethylaminuria (TMAuria), an inborn error of metabolism associated with a fishy body odor emitting from sweat, urine and breath. Genetic mutations in FMO3 lead to the N-oxidation of amino-trimethylamine derived from food products, thus producing the malodor associated with TMAuria.

Supplier: Bioss

The Flavin containing monooxygenase family consists of five gene products, FMO1-5, that are major enzymatic oxidants involved in the metabolism of various therapeutics. Located in the liver, FMO3 is a hepatic microsomal enzyme that oxygenates soft nucleophiles such as secondary and tertiary amines. Through its N-oxygenase capabilities, FMO3 acts on a variety of xenobiotics to catalyze oxidative digestion. Defects in the FMO3 gene are the primary cause of trimethylaminuria (TMAuria), an inborn error of metabolism associated with a fishy body odor emitting from sweat, urine and breath. Genetic mutations in FMO3 lead to the N-oxidation of amino-trimethylamine derived from food products, thus producing the malodor associated with TMAuria.

Supplier: Bioss

The Flavin containing monooxygenase family consists of five gene products, FMO1-5, that are major enzymatic oxidants involved in the metabolism of various therapeutics. Located in the liver, FMO3 is a hepatic microsomal enzyme that oxygenates soft nucleophiles such as secondary and tertiary amines. Through its N-oxygenase capabilities, FMO3 acts on a variety of xenobiotics to catalyze oxidative digestion. Defects in the FMO3 gene are the primary cause of trimethylaminuria (TMAuria), an inborn error of metabolism associated with a fishy body odor emitting from sweat, urine and breath. Genetic mutations in FMO3 lead to the N-oxidation of amino-trimethylamine derived from food products, thus producing the malodor associated with TMAuria.

Supplier: Bioss

The Flavin containing monooxygenase family consists of five gene products, FMO1-5, that are major enzymatic oxidants involved in the metabolism of various therapeutics. Located in the liver, FMO3 is a hepatic microsomal enzyme that oxygenates soft nucleophiles such as secondary and tertiary amines. Through its N-oxygenase capabilities, FMO3 acts on a variety of xenobiotics to catalyze oxidative digestion. Defects in the FMO3 gene are the primary cause of trimethylaminuria (TMAuria), an inborn error of metabolism associated with a fishy body odor emitting from sweat, urine and breath. Genetic mutations in FMO3 lead to the N-oxidation of amino-trimethylamine derived from food products, thus producing the malodor associated with TMAuria.